Exposure to in-utero hyperglycaemia influences later cardiometabolic risk, although few studies include women with type 2 diabetes (T2D) or assess maternal body mass index (BMI) as a potential confounder. This study explored the association of maternal T2D and gestational diabetes mellitus (GDM) with childhood anthropometry, and the influence of maternal BMI on these associations.
The PANDORA birth cohort comprises 1138 women and 1163 children, women with GDM and T2D recruited from a hyperglycaemia in pregnancy register, and women with normoglycaemia from the community. Wave 1 follow-up included 423 children, aged 1.5-5 years (median 2.5 years). Multivariable linear regression assessed associations between maternal antenatal variables with offspring anthropometry (weight, height, BMI, skinfold thicknesses, waist, arm and head circumferences).
Greater maternal BMI was associated with increased anthropometric measures in offspring independent of maternal glycaemic status. After adjustment, including for maternal BMI, children exposed to GDM had lower mean weight (-0.54kg, 95% CI -0.99, -0.11), BMI (-0.55kg/m2, 95% CI -0.91, -0.20), head (-0.52cm, 95% CI -0.88, -0.16) and mid-upper arm (-0.32cm, 95% CI -0.63, -0.01) circumferences, and greater mean suprailiac skinfold (0.78mm, 95% CI 0.13, 1.43), compared to children exposed to normoglycaemia. Children exposed to T2D had smaller mean head circumference (-0.82cm, 95% CI -1.33, -0.31) than children exposed to normoglycaemia. Adjustment for maternal BMI strengthened the negative association between GDM and child weight, BMI and circumferences. Maternal T2D was no longer associated with greater mean skinfolds (p=0.14) and waist circumference (p=0.18) in children after adjustment for maternal BMI.
Compared to children exposed to normoglycaemia, children exposed to GDM had greater suprailiac skinfold thickness, despite having lower mean weight, BMI and mid-upper arm circumference, and both GDM and T2D were associated with smaller head circumference. Future research should assess whether childhood anthropometric differences influence lifetime cardiometabolic and neurodevelopmental risk.